CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases

CaV2.1 (P/Q type) Ca2+ channels have a fundamental role mediating fast transmitter release at central and peripheral synaptic terminals. Various neurological diseases have been attributed to genetic and autoimmune malfunctioning of P/Q channels, including ataxia, migraine and myasthenic syndromes. T...

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Detalles Bibliográficos
Autor principal: Uchitel, O.D.
Formato: CHAP
Materias:
Ataxia
CaV2.1 subunits
Channelopathy
Migraine
Myasthenic syndromes
P/Q type current
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_97894007_v9789400763340_n_p263_Uchitel
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires
id todo:paper_97894007_v9789400763340_n_p263_Uchitel
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spelling todo:paper_97894007_v9789400763340_n_p263_Uchitel2023-10-03T16:45:19Z CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases Uchitel, O.D. Ataxia CaV2.1 subunits Channelopathy Migraine Myasthenic syndromes P/Q type current CaV2.1 (P/Q type) Ca2+ channels have a fundamental role mediating fast transmitter release at central and peripheral synaptic terminals. Various neurological diseases have been attributed to genetic and autoimmune malfunctioning of P/Q channels, including ataxia, migraine and myasthenic syndromes. This chapter focuses on recent advances on the understanding of the pathogenic mechanisms underlying these disorders. © Springer Science+Business Media Dordrecht 2013. All rights are reserved. Fil:Uchitel, O.D. Universidad de Buenos Aires. Facultad de Ciencias Exactas y Naturales; Argentina. CHAP info:eu-repo/semantics/openAccess http://creativecommons.org/licenses/by/2.5/ar http://hdl.handle.net/20.500.12110/paper_97894007_v9789400763340_n_p263_Uchitel
institution Universidad de Buenos Aires
institution_str I-28
repository_str R-134
collection Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA)
topic Ataxia
CaV2.1 subunits
Channelopathy
Migraine
Myasthenic syndromes
P/Q type current
spellingShingle Ataxia
CaV2.1 subunits
Channelopathy
Migraine
Myasthenic syndromes
P/Q type current
Uchitel, O.D.
CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
topic_facet Ataxia
CaV2.1 subunits
Channelopathy
Migraine
Myasthenic syndromes
P/Q type current
description CaV2.1 (P/Q type) Ca2+ channels have a fundamental role mediating fast transmitter release at central and peripheral synaptic terminals. Various neurological diseases have been attributed to genetic and autoimmune malfunctioning of P/Q channels, including ataxia, migraine and myasthenic syndromes. This chapter focuses on recent advances on the understanding of the pathogenic mechanisms underlying these disorders. © Springer Science+Business Media Dordrecht 2013. All rights are reserved.
format CHAP
author Uchitel, O.D.
author_facet Uchitel, O.D.
author_sort Uchitel, O.D.
title CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
title_short CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
title_full CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
title_fullStr CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
title_full_unstemmed CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
title_sort cav2.1 (p/q) voltage activated ca2+ channels and synaptic transmission in genetic and autoimmune diseases
url http://hdl.handle.net/20.500.12110/paper_97894007_v9789400763340_n_p263_Uchitel
work_keys_str_mv AT uchitelod cav21pqvoltageactivatedca2channelsandsynaptictransmissioningeneticandautoimmunediseases
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