Juvenile exposure to a high fat diet promotes behavioral and limbic alterations in the absence of obesity

The incidence of metabolic disorders including obesity, type 2 diabetes and metabolic syndrome have seriously increased in the last decades. These diseases – with growing impact in modern societies – constitute major risk factors for neurodegenerative disorders such as Alzheimer's disease (AD),...

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Detalles Bibliográficos
Autores principales: Vinuesa, A., Pomilio, C., Menafra, M., Bonaventura, M.M., Garay, L., Mercogliano, M.F., Schillaci, R., Lux Lantos, V., Brites, F., Beauquis, J., Saravia, F.
Formato: JOUR
Materias:
Cognitive performance
Emotionality
High fat diet
Hippocampal neuroinflammation
Neurogenesis
Periadolescence
insulin receptor substrate 1
insulin receptor substrate 2
interleukin 1beta
Ki 67 antigen
phospholipase A2
protein c fos
protein kinase B
tumor necrosis factor alpha
adult
amygdala
animal experiment
animal model
animal tissue
anxiety
Article
behavior change
cell proliferation
controlled study
dentate gyrus
depression
disease association
dyslipidemia
enzyme activity
enzyme phosphorylation
gene expression
insulin resistance
lipid composition
lipid diet
male
memory disorder
motivation
mouse
nervous system inflammation
nonhuman
obesity
population exposure
priority journal
protein blood level
protein expression
protein phosphorylation
short term memory
spatial memory
animal
animal behavior
C57BL mouse
cognitive defect
hippocampus
hyperinsulinism
inflammation
metabolism
nervous system development
pathology
pathophysiology
physiology
Amygdala
Animals
Behavior, Animal
Cognitive Dysfunction
Diet, High-Fat
Dyslipidemias
Hippocampus
Hyperinsulinism
Inflammation
Male
Mice
Mice, Inbred C57BL
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_03064530_v72_n_p22_Vinuesa
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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