Central pontlne myelinolysis induced by hyperemesis gravidarum
An 18-vear-old woman in her first pregnancy with hyperemesis gravidarum, presented dehydration, without hyponatremia. She was confused with profound disorientation, apathy, and drowsiness. She presented upbeating nistagmus on upward gaze and gate ataxia recognised as Wernicke's encephalopathy....
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1999
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| Acceso en línea: | https://bibliotecadigital.exactas.uba.ar/collection/paper/document/paper_00257680_v59_n2_p176_Tonelli http://hdl.handle.net/20.500.12110/paper_00257680_v59_n2_p176_Tonelli |
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paper:paper_00257680_v59_n2_p176_Tonelli2023-06-08T14:53:32Z Central pontlne myelinolysis induced by hyperemesis gravidarum Central pontine myelinolysis Hyperemesis gravidarum Hyponatremia Wernicke's encephalopathy adolescent article case report central pontine myelinolysis female human hyperemesis gravidarum hypernatremia male pregnancy Adolescent Female Humans Hyperemesis Gravidarum Hypernatremia Male Myelinolysis, Central Pontine Pregnancy An 18-vear-old woman in her first pregnancy with hyperemesis gravidarum, presented dehydration, without hyponatremia. She was confused with profound disorientation, apathy, and drowsiness. She presented upbeating nistagmus on upward gaze and gate ataxia recognised as Wernicke's encephalopathy. Laboratory tests demonstrated hypokalemia, hypernatremia and aminotransferase elevation. The serum osmolality was 319 mOsm/kg and the water deficiency 2.73 I. The patient developed weakness in the four limbs, with hypotonicity, absence of tendon reflexes and showed bilateral Babinski signs. A T2 weighted sagittal cranial-magnetic resonance imaging revealed a high signal within mid-pons suggesting central pontine myelinolysis. In this case we highlight the absence of hyponatremia. Furthermore, the central pontine myelinolysis was probably secondary to hypokalemia, hypernatremia and hyperosmolality. 1999 https://bibliotecadigital.exactas.uba.ar/collection/paper/document/paper_00257680_v59_n2_p176_Tonelli http://hdl.handle.net/20.500.12110/paper_00257680_v59_n2_p176_Tonelli |
| institution |
Universidad de Buenos Aires |
| institution_str |
I-28 |
| repository_str |
R-134 |
| collection |
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) |
| topic |
Central pontine myelinolysis Hyperemesis gravidarum Hyponatremia Wernicke's encephalopathy adolescent article case report central pontine myelinolysis female human hyperemesis gravidarum hypernatremia male pregnancy Adolescent Female Humans Hyperemesis Gravidarum Hypernatremia Male Myelinolysis, Central Pontine Pregnancy |
| spellingShingle |
Central pontine myelinolysis Hyperemesis gravidarum Hyponatremia Wernicke's encephalopathy adolescent article case report central pontine myelinolysis female human hyperemesis gravidarum hypernatremia male pregnancy Adolescent Female Humans Hyperemesis Gravidarum Hypernatremia Male Myelinolysis, Central Pontine Pregnancy Central pontlne myelinolysis induced by hyperemesis gravidarum |
| topic_facet |
Central pontine myelinolysis Hyperemesis gravidarum Hyponatremia Wernicke's encephalopathy adolescent article case report central pontine myelinolysis female human hyperemesis gravidarum hypernatremia male pregnancy Adolescent Female Humans Hyperemesis Gravidarum Hypernatremia Male Myelinolysis, Central Pontine Pregnancy |
| description |
An 18-vear-old woman in her first pregnancy with hyperemesis gravidarum, presented dehydration, without hyponatremia. She was confused with profound disorientation, apathy, and drowsiness. She presented upbeating nistagmus on upward gaze and gate ataxia recognised as Wernicke's encephalopathy. Laboratory tests demonstrated hypokalemia, hypernatremia and aminotransferase elevation. The serum osmolality was 319 mOsm/kg and the water deficiency 2.73 I. The patient developed weakness in the four limbs, with hypotonicity, absence of tendon reflexes and showed bilateral Babinski signs. A T2 weighted sagittal cranial-magnetic resonance imaging revealed a high signal within mid-pons suggesting central pontine myelinolysis. In this case we highlight the absence of hyponatremia. Furthermore, the central pontine myelinolysis was probably secondary to hypokalemia, hypernatremia and hyperosmolality. |
| title |
Central pontlne myelinolysis induced by hyperemesis gravidarum |
| title_short |
Central pontlne myelinolysis induced by hyperemesis gravidarum |
| title_full |
Central pontlne myelinolysis induced by hyperemesis gravidarum |
| title_fullStr |
Central pontlne myelinolysis induced by hyperemesis gravidarum |
| title_full_unstemmed |
Central pontlne myelinolysis induced by hyperemesis gravidarum |
| title_sort |
central pontlne myelinolysis induced by hyperemesis gravidarum |
| publishDate |
1999 |
| url |
https://bibliotecadigital.exactas.uba.ar/collection/paper/document/paper_00257680_v59_n2_p176_Tonelli http://hdl.handle.net/20.500.12110/paper_00257680_v59_n2_p176_Tonelli |
| _version_ |
1768546289300013056 |