Protective action of erythropoietin on neuronal damage induced by activated microglia

Inflammation is a physiological defense response, but may also represent a potential pathological process in neurological diseases. In this regard, microglia have a crucial role in either progression or amelioration of degenerative neuronal damage. Because of the role of hypoxia in pro-inflammatory...

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Detalles Bibliográficos
Autores principales: Wenker, S.D., Chamorro, M.E., Vittori, D.C., Nesse, A.B.
Formato: JOUR
Materias:
erythropoietin
hypoxia
inflammation
microglia
neuroprotection
cobalt chloride
cycline
inducible nitric oxide synthase
nitrite
reactive oxygen metabolite
tumor necrosis factor alpha
animal cell
animal experiment
animal model
antiinflammatory activity
antioxidant activity
apoptosis
article
cell activation
cell proliferation
cell survival
controlled study
cytokine release
human
human cell
macrophage
mouse
nerve cell
nerve cell lesion
nonhuman
oxidative stress
priority journal
protein expression
upregulation
Animals
Cell Hypoxia
Cell Proliferation
Cells, Cultured
Cobalt
Culture Media, Conditioned
Erythropoietin
Humans
Inflammation
Mice
Microglia
Neurons
Neuroprotective Agents
Neurotoxicity Syndromes
Nitric Oxide Synthase Type II
Nitrites
Proliferating Cell Nuclear Antigen
Reactive Oxygen Species
Receptors, Erythropoietin
Tumor Necrosis Factor-alpha
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_1742464X_v280_n7_p1630_Wenker
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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