Untangling Galectin-Driven Regulatory Circuits in Autoimmune Inflammation

Although progress has been made in understanding the mechanisms implicated in the pathogenesis of autoimmune inflammation, studies aimed at identifying the mediators of these pathways will be necessary to develop more selective therapies. Galectins, a family of glycan-binding proteins, play central...

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Detalles Bibliográficos
Autores principales: Toscano, M.A., Martínez Allo, V.C., Cutine, A.M., Rabinovich, G.A., Mariño, K.V.
Formato: JOUR
Materias:
autoimmunity
chronic inflammation
galectins
glycans
immunoregulation
ecalectin
galectin
galectin 2
galectin 3
gamma interferon
glycan
interleukin 10
interleukin 17
interleukin 1beta
interleukin 27
tumor necrosis factor
biological factor
apoptosis
astrocyte
autoimmune disease
CD4+ T lymphocyte
CD8+ T lymphocyte
collagen-induced arthritis
Crohn disease
dendritic cell
endothelium cell
experimental autoimmune encephalomyelitis
glycosylation
human
immunocompetent cell
immunological tolerance
immunomodulation
inflammation
inflammatory bowel disease
insulin dependent diabetes mellitus
intestine flora
lymphocytic infiltration
macrophage
microglia
multiple sclerosis
nervous system inflammation
nonhuman
protein carbohydrate interaction
protein expression
protein function
regulatory T lymphocyte
Review
rheumatoid arthritis
systemic lupus erythematosus
Th1 cell
Th17 cell
ulcerative colitis
upregulation
adverse drug reaction
animal
drug effect
metabolism
Animals
Autoimmune Diseases
Autoimmunity
Biological Factors
Drug-Related Side Effects and Adverse Reactions
Galectins
Humans
Inflammation
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_14714914_v24_n4_p348_Toscano
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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