Hexachlorobenzene triggers apoptosis in rat thyroid follicular cells

Hexachlorobenzene (HCB) is a widespread environmental pollutant. Chronic exposure of humans to HCB produces a number of effects, such as triggering of porphyria, increased synthesis of liver microsomal enzymes, neurological symptoms, immunological disorders and thyroid dysfunctions. In rats, HCB ind...

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Detalles Bibliográficos
Autores principales: Chiappini, F., Alvarez, L., Lux-Lantos, V., Randi, A.S., de Pisarev, D.L.K.
Formato: JOUR
Materias:
Apoptosis
Hexachlorobenzene
Rat thyroid
caspase 9
cytochrome c
DNA nucleotidylexotransferase
hexachlorobenzene
messenger RNA
procaspase 8
thyroid hormone
thyroxine
transforming growth factor beta1
animal cell
animal experiment
animal tissue
apoptosis
article
cell count
cell proliferation
controlled study
DNA fragmentation
female
gene expression
growth regulation
nick end labeling
nonhuman
protein secretion
rat
regulatory mechanism
reverse transcription polymerase chain reaction
thyroid follicle cell
thyroid hormone blood level
thyroid weight
tissue structure
upregulation
Animals
Blotting, Western
Caspase 8
Caspase 9
Cell Proliferation
DNA Fragmentation
Endocrine Disruptors
Environmental Pollutants
Enzyme Induction
Female
In Situ Nick-End Labeling
Mitochondria
Organ Size
Rats
Rats, Wistar
Reverse Transcriptase Polymerase Chain Reaction
RNA, Messenger
Signal Transduction
Subcellular Fractions
Thyroid Gland
Thyroid Hormones
Transforming Growth Factor beta1
Animalia
Rattus
Rattus norvegicus
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_10966080_v108_n2_p301_Chiappini
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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