Heme oxygenase 1 (HO-1) challenges the angiogenic switch in prostate cancer

Prostate cancer (PCa) is the second leading cause of cancer-associated death in men. Once a tumor is established it may attain further characteristics via mutations or hypoxia, which stimulate new blood vessels. Angiogenesis is a hallmark in the pathogenesis of cancer and inflammatory diseases that...

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Detalles Bibliográficos
Autores principales: Ferrando, M., Gueron, G., Elguero, B., Giudice, J., Salles, A., Leskow, F.C., Jares-Erijman, E.A., Colombo, L., Meiss, R., Navone, N., De Siervi, A., Vazquez, E.
Formato: JOUR
Materias:
Angiogenesis
Heme oxygenase 1 (HO-1)
NF-κB
Prostate cancer
VEGF
CD34 antigen
gelatinase B
heme oxygenase 1
hypoxia inducible factor 1alpha
immunoglobulin enhancer binding protein
reactive oxygen metabolite
vasculotropin A
vasculotropin C
vasculotropin receptor 2
very late activation antigen 5
animal experiment
animal model
animal tissue
article
controlled study
gene expression regulation
gene overexpression
genetic transfection
heme oxygenase 1 gene
in vivo study
male
microvasculature
mouse
nonhuman
oxidative stress
priority journal
prostate cancer
receptor down regulation
signal transduction
transcription regulation
tumor gene
tumor vascularization
Analysis of Variance
Animals
DNA Primers
Gene Expression Regulation, Neoplastic
Heme Oxygenase-1
Histological Techniques
Humans
Immunohistochemistry
Luciferases
Male
Mice
Mice, Nude
Neovascularization, Pathologic
Plasmids
Prostatic Neoplasms
Reactive Oxygen Species
Reverse Transcriptase Polymerase Chain Reaction
Vascular Endothelial Growth Factor A
Vascular Endothelial Growth Factor C
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_09696970_v14_n4_p467_Ferrando
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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