Transrepression of NF-κB is not required for glucocorticoid-mediated protection of TNF-α-induced apoptosis on fibroblasts

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The cellular resistance to tumor necrosis factor (TNF) of most cell types has been attributed to both a protective pathway induced by this cytokine and the preexistence of protective factors in the target cell. NF-κB has been postulated as one of the principal factors involved in antiapoptotic gene...

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Detalles Bibliográficos
Autores principales: Costas, M.A., Müller Igaz, L., Holsboer, F., Arzt, E.
Formato: Artículo publishedVersion
Lenguaje:Inglés
Publicado: 2000
Materias:
Apoptosis
Glucocorticoid
Glucocorticoid receptor
Nuclear factor-κB
Tumor necrosis factor
beta galactosidase
dexamethasone
glucocorticoid
glucocorticoid receptor
I kappa B
immunoglobulin enhancer binding protein
luciferase
messenger RNA
tumor necrosis factor alpha
tumor necrosis factor alpha receptor
DNA binding protein
NF kappaB inhibitor alpha
NF-kappaB inhibitor alpha
animal cell
apoptosis
article
cell protection
controlled study
cytotoxicity
fibroblast
gene expression regulation
gene repression
mouse
nonhuman
priority journal
protein expression
suppressor cell
target cell destruction
transactivation
animal
biosynthesis
cell line
drug antagonism
genetic transfection
metabolism
Animals
Cell Line
DNA-Binding Proteins
Fibroblasts
Glucocorticoids
I-kappa B Proteins
Mice
NF-kappa B
Receptors, Glucocorticoid
Transfection
Tumor Necrosis Factor-alpha
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_01674889_v1499_n1-2_p122_Costas
Aporte de:
Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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