CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
CaV2.1 (P/Q type) Ca2+ channels have a fundamental role mediating fast transmitter release at central and peripheral synaptic terminals. Various neurological diseases have been attributed to genetic and autoimmune malfunctioning of P/Q channels, including ataxia, migraine and myasthenic syndromes. T...
Guardado en:
Autor principal: | Uchitel, Osvaldo Daniel |
---|---|
Publicado: |
2013
|
Materias: | |
Acceso en línea: | https://bibliotecadigital.exactas.uba.ar/collection/paper/document/paper_97894007_v9789400763340_n_p263_Uchitel http://hdl.handle.net/20.500.12110/paper_97894007_v9789400763340_n_p263_Uchitel |
Aporte de: |
Ejemplares similares
-
CaV2.1 (P/Q) voltage activated Ca2+ channels and synaptic transmission in genetic and autoimmune diseases
por: Uchitel, O.D. -
Ca V2.1 voltage activated calcium channels and synaptic transmission in familial hemiplegic migraine pathogenesis
por: Uchitel, Osvaldo Daniel, et al.
Publicado: (2012) -
Ca V2.1 voltage activated calcium channels and synaptic transmission in familial hemiplegic migraine pathogenesis
por: Uchitel, O.D., et al. -
Calcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models
por: Uchitel, O.D., et al. -
Constitutive activity of the Ghrelin receptor reduces surface expression of voltage-gated Ca2+ channels in a CaVβ-dependent manner
por: Mustafá, Emilio Román, et al.
Publicado: (2017)